Review ArticlesThe Probable Mechanism of Traumatic Angle Recession and CyclodialysisPujari, Amar MD; Selvan, Harathy MD; Behera, Aswini Kumar MD; Gagrani, Meghal MD; Kapoor, Saloni MBBS; Dada, Tanuj MDAuthor Information Dr. Rajendra Prasad Centre for Ophthalmic Sciences, All India Institute of Medical Sciences, New Delhi, India The authors declare no conflict of interest. Reprints: Amar Pujari, MD, Dr. Rajendra Prasad Centre for Ophthalmic Sciences, All India Institute of Medical Sciences, Room No 212, Second Floor, RPC-1, New Delhi 110029, India (e-mail: firstname.lastname@example.org). Received April 29, 2019 Accepted August 15, 2019 Online date: August 26, 2019 Journal of Glaucoma: January 2020 - Volume 29 - Issue 1 - p 67–70 doi: 10.1097/IJG.0000000000001358 Buy Metrics Abstract During blunt ocular trauma, the anteroposterior compressive forces confronted lead to consequent equatorial expansion of the globe. This may result in ciliary body trauma, typically manifesting as angle recession or cyclodialysis. The authors hypothesize that a likely asymmetric contraction between the longitudinal and circular ciliary fibers, and an intrinsic weak “oblique buffer zone” creates a plane of separation between the 2, resulting in angle recession. When stronger forces are met with, the equatorial expansion of the sclera may outperform the ability of the ciliary body to follow it, and the taut longitudinal ciliary fibers may subsequently disinsert from the scleral spur causing cyclodialysis. In addition to this, the routinely thought dismembering aqueous jets directed toward the angle may also accentuate ciliary body trauma. Therefore, the vivid distractive external forces along with the complex ciliary muscle anatomy and differential functionality may play a crucial role in causation of post-traumatic angle recession and cyclodialysis. Copyright © 2020 Wolters Kluwer Health, Inc. All rights reserved.