ZIP12 Contributes to Hypoxic Pulmonary Hypertension by Driving Phenotypic Switching of Pulmonary Artery Smooth Muscle Cells: Erratum

In the February 2022 issue of the Journal of Cardiovascular Pharmacology, in the article by Zhu et al ¹, “ZIP12 Contributes to Hypoxic Pulmonary Hypertension by Driving Phenotypic Switching of Pulmonary Artery Smooth Muscle Cells, there are errors in Figures 4D and 4E.”

The authors regret the β-actin western blot images in Figures 4D and 4E were duplicated when combining the results together (Figure 4D had its own β-actin). Below is revised Figure 4 in which β-actin in Figure 4D has been changed. The authors apologize for the error.

FIGURE 4.: ZIP12 promoted proliferation and phenotypic switch of PASMCs. A, B, ZIP12 mRNA and protein expression in the PASMCs; (C) effect of ZIP12 siRNA on proliferation of PASMCs treated with hypoxia measured by EdU, scale bar: 100 mm; (D–F) effect of ZIP12 siRNA on the a-SMA, vimentin, and OPN expression of PASMCs exposed to hypoxia. All values are expressed as mean 6 SEM. n = 6/4, *P ˂ 0.05, **P ˂ 0.01 versus normoxia or +NC.