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Erratum

ZIP12 Contributes to Hypoxic Pulmonary Hypertension by Driving Phenotypic Switching of Pulmonary Artery Smooth Muscle Cells: Erratum

Journal of Cardiovascular Pharmacology: May 2022 - Volume 79 - Issue 5 - p 758
doi: 10.1097/FJC.0000000000001266
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In the February 2022 issue of the Journal of Cardiovascular Pharmacology, in the article by Zhu et al 1, “ZIP12 Contributes to Hypoxic Pulmonary Hypertension by Driving Phenotypic Switching of Pulmonary Artery Smooth Muscle Cells, there are errors in Figures 4D and 4E.”

The authors regret the β-actin western blot images in Figures 4D and 4E were duplicated when combining the results together (Figure 4D had its own β-actin). Below is revised Figure 4 in which β-actin in Figure 4D has been changed. The authors apologize for the error.

F1
FIGURE 4.:
ZIP12 promoted proliferation and phenotypic switch of PASMCs. A, B, ZIP12 mRNA and protein expression in the PASMCs; (C) effect of ZIP12 siRNA on proliferation of PASMCs treated with hypoxia measured by EdU, scale bar: 100 mm; (D–F) effect of ZIP12 siRNA on the a-SMA, vimentin, and OPN expression of PASMCs exposed to hypoxia. All values are expressed as mean 6 SEM. n = 6/4, *P ˂ 0.05, **P ˂ 0.01 versus normoxia or +NC.

Reference

1. Zhu T, Wang X, Zheng Z, et al. ZIP12 Contributes to Hypoxic Pulmonary Hypertension by Driving Phenotypic Switching of Pulmonary Artery Smooth Muscle Cells. J Cardiovasc Pharmacol 2022;79:235-243.
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