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Correction to: Demystifying Esophageal Lichen Planus: A Comprehensive Review of a Rare Disease You Will See in Practice

Jacobs, John W. Jr MD1; Kukreja, Keshav MD1; Camisa, Charles MD2; Richter, Joel E. MD1

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The American Journal of Gastroenterology: June 2022 - Volume 117 - Issue 6 - p 1016-1017
doi: 10.14309/ajg.0000000000001710
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Correction to:The American Journal of Gastroenterology: January 2022; 117(1):70–77. doi:

In the January 2022 issue of The American Journal of Gastroenterology, “Demystifying Esophageal Lichen Planus: A Comprehensive Review of a Rare Disease You Will See in Practice,” the images were to be published in color. The color figures appear below.

Figure 1.:
Oral manifestations of lichen planus. Careful examination of the oral cavity can show desquamative gingivitis with the classically described beefy-red gums (a), aphthous-appearing ulcerations (b), and a white, lacy, reticular pattern on the buccal mucosa (c).
Figure 2.:
Extraesophageal manifestations of lichen planus. On the skin, purple, irregular, raised papules can be seen (a). Lichen planus lesions on an African American patient healing with postinflammatory hyperpigmentation (b). Toenail involvement demonstrates atrophy of the nail plates with rough surfaces and longitudinal ridges (c). This active plaque of lichen planus shows abundant white Wickham striae on the surface and healing areas with postinflammatory hyperpigmentation (d).
Figure 3.:
Classic endoscopic findings of esophageal lichen planus: subtle findings can include pale (a) and edematous mucosa (b). Because of disruption of the basement membrane, passage of the endoscope alone can cause mucosal peeling (c, d). A thick white exudate (e, f) and esophageal strictures, especially in the upper midesophagus (e, f), can be seen.
Figure 4.:
High-power view of histopathology of the basal epithelium in oral lichen planus shows lymphocytic infiltrate in subepithelial tissue. The pink globule, marked by the larger horizontal black arrow, is the Civatte body, a characteristic sign of lichen planus, and represents degenerating basal cells (smaller vertical black arrows) caused by autoimmune T-cell–mediated damage. Stained with H&E, magnification 340. Courtesy of Carl M. Allen, DDS, MSD.
Figure 5.:
Progression of esophageal lichen planus to esophageal carcinoma rapidly, with 2 endoscopies only 6 months apart. The former endoscopy (a, b) showed moderate exudate and a narrowing in the midesophagus, which had been previously dilated. Follow-up endoscopy 6 months later showed a new mass in the midesophagus (d). Otherwise, the proximal and distal parts of the esophagus were rather unremarkable compared with previous endoscopies (c).
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