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Maximal Lactate Steady State in Patients with Coronary Heart Disease in Contrast to Healthy Subjects: 2538Board#46 2:00 PM – 3:00 PM

Pokan, Rochus FACSM; Primus, Alfred; Hofmann, Peter; Von Duvillard, Serge FACSM; Wonisch, Manfred; Smekal, Gerhard; Bachl, Norbert; Schmid, Peter

Medicine & Science in Sports & Exercise: May 2006 - Volume 38 - Issue 5 - p S477
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1Univ. of Vienna, Austria, Vienna, Austria.

2Univ. of Graz, Austria, Graz, Austria.

3Texas A&M University-Commerce, Texas USA, Vienna, TX.

4Center for Cardiac Rehabilitation, Austria, Bad Schallerbach, Austria.

Email: rochus.pokan@univie.ac.at

PURPOSE: Maximal lactate steady state (MLSS) presumably corresponds to the highest constant work load that can be performed by oxidative metabolism. The aim of this study was to investigate the MLSS in female and male patients with coronary heart disease (CHD) in contrast to healthy subjects (HS) of comparable age and physical education students (PES).

METHODS: Incremental and constant work load tests were performed in patients with coronary heart disease (CHD), healthy subjects (HS) and physical education students (PES). Each subject performed an incremental test on a cycle ergometer in an upright position to the limit of tolerance at an initial level of 20 Watt (W) and increments of 10 W, 15 W, 20 W and 25 W (adjusted for individual functional capacity), added every min to the limit of tolerance. LTP2 was denned as the second abrupt increase in lactate concentration (LA). Each subject performed at least two MLSS tests. As in the incremental test, workload was increased every min until power output at LTP2 was obtained. Steady state workload was kept constant for an additional 20 min and monitored via LA. If LA increased more than 10% of L Amax mmol.l-1during the last 10 min, an additional test of 10 W, 15 W, 20 W or 25 W load-decrease was conducted. If LA remained constant or decreased, power output was increased by 10 W, 15 W, 20 W or 25 W.

RESULTS:

Table

Table

CONCLUSIONS: The relative large +SD in LA values are independent of age, sex, or coronary disease status. Therefore, individual lactate measures have no value for training regulation

© 2006 American College of Sports Medicine