H-13H Free Communication/Thematic Poster Winter Sports
When exposed to a cold environment, peripheral blood flow decreases in an effort to maintain core body temperature and to protect pulmonary tissues. This change in blood flow increases central venous pressure as well as tracheal and bronchial blood flow. It has been demonstrated that inspiring cold air can stimulate endothelial cells of the heart and pulmonary trunk to release endothelin, a powerful vasoconstrictor creating conflicting responses between the central and peripheral factors. The PURPOSE of the study was to determine if a heat exchange face mask (FM) could maintain peripheral blood flow and reduce the influence of endothelin simply by warming and humidifying inspired air.
Five healthy subjects participated in this repeated measures, cross-over design study. They were seated in a −16°C environment for 60 minutes while completing FM and no face mask (NFM) trials. Subjects were dressed in the same insulated clothes for both trials.
During the FM trial HR, Systolic BP, MAP, PV, and Endothelin all remained unchanged from baseline. However, during the NFM trial HR decreased significantly (p < 0.05) and Systolic BP, MAP and Endothelin all increased, (p> < 0.05) while no significant difference was noted for % change in PV.
It can be concluded that the heat exchange mask aids in reducing endothelin-induced cardiovascular stress compared to no intervention during 60 minutes of cold exposure. Supported by Polar Wrap, Inc.