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Alzheimer Disease: Current Topics

Creator:   Dr. Raymond A. Sobel, Editor-in-Chief
Created:   5/5/2010
Contains:  36 items
This collection features clinical and research articles on Alzheimer disease published in JNEN from 2008 through 2010.

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Thinking Outside the Box: Alzheimer-Type Neuropathology That Does Not Map Directly Onto Current Consensus Recommendations

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Amyloid Deposition and Influx Transporter Expression at the Blood-Brain Barrier Increase in Normal Aging

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Paxillin and Hydrogen Peroxide-Inducible Clone 5 Expression and Distribution in Control and Alzheimer Disease Hippocampi

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Curcumin Labeling of Neuronal Fibrillar Tau Inclusions in Human Brain Samples

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Genetic Ablation of Luteinizing Hormone Receptor Improves the Amyloid Pathology in a Mouse Model of Alzheimer Disease

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Oxidative Stress in the Progression of Alzheimer Disease in the Frontal Cortex

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Clinical, Neuropathologic, and Biochemical Profile of the Amyloid Precursor Protein I716F Mutation

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Caspase-6 Activation in Familial Alzheimer Disease Brains Carrying Amyloid Precursor Protein or Presenilin I or Presenilin II Mutations

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Caspase-6 Activation in Familial Alzheimer Disease Brains Carrying Amyloid Precursor Protein or Presenilin I or Presenilin II Mutations: Erratum

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Neuropathology and Cognitive Impairment in Alzheimer Disease: A Complex but Coherent Relationship

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Statins Reduce the Neurofibrillary Tangle Burden in a Mouse Model of Tauopathy

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Sirtuin 1 Reduction Parallels the Accumulation of Tau in Alzheimer Disease

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DNA Methylation of Alzheimer Disease and Tauopathy-Related Genes in Postmortem Brain

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Amyloid β-Induced Nerve Growth Factor Dysmetabolism in Alzheimer Disease

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Neprilysin and Insulin-Degrading Enzyme Levels Are Increased in Alzheimer Disease in Relation to Disease Severity

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Tangle-Bearing Neurons Survive Despite Disruption of Membrane Integrity in a Mouse Model of Tauopathy

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Increased Matrix Metalloproteinase 9 Activity in Mild Cognitive Impairment

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Brains With Medial Temporal Lobe Neurofibrillary Tangles But No Neuritic Amyloid Plaques Are a Diagnostic Dilemma But May Have Pathogenetic Aspects Distinct From Alzheimer Disease

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Increased Association Between Rough Endoplasmic Reticulum Membranes and Mitochondria in Transgenic Mice That Express P301L Tau

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Cerebrovascular Smooth Muscle Actin Is Increased in Nondemented Subjects With Frequent Senile Plaques at Autopsy: Implications for the Pathogenesis of Alzheimer Disease

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