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The Metabolic Response to Acute Traumatic Brain Injury and Implications for Nutritional Support

Pepe, Judith L. MD, FACS; Barba, Carlos A. MD, FRCSC, FACS

Journal of Head Trauma Rehabilitation:
Current Trends and Technologies in Dysphagia Management
Abstract

An overview of the metabolic response to acute traumatic brain injury is presented. The consequences of hypermetabolism, hypercatabolism, and an altered immune function are discussed. Once a person with acute traumatic brain injury develops this hyperdynamic state, the resultant excessive protein breakdown ensues. This can lead to malnutrition. The feeding methods used to prevent malnutrition are discussed, along with the proper alimentation to provide to diminish the hyperdynamic state and improve immune function.

Author Information

Director Surgical Critical Care, Associate Director Trauma, Director Nutritional Support, Saint Francis Hospital and Medical Center, Associate Professor of Surgery, University of Connecticut, Hartford, Connecticut (Pepe)

Associate Director of Trauma and Surgical Critical Care, Saint Francis Hospital and Medical Center, Assistant Professor of Surgery, University of Connecticut, Hartford, Connecticut (Barba)

Address correspondence to: J. Pepe, Surgical Critical Care, Saint Francis Hospital and Medical Center, 114 Woodland Street, Hartford, CT 06105.

© 1999 Lippincott Williams & Wilkins, Inc.